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Current clinical data indicate that GLP-1R agonists reduce BP in diabetic patients (92) and the antihypertensive effect can also be observed in Dahl salt-sensitive rats (93), in Ang II -induced hypertension rats (91) and SHRs rats (94)
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The glucagon component appears to preferentially direct energy expenditure toward fat oxidation rather than muscle catabolism, especially when paired with even moderate resistance training
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