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Preclinical and in vitro work suggests that GHK-Cu can modulate collagen and glycosaminoglycan turnover, influence fibroblast and immune-cell behavior, and interact with pathways related to oxidative stress and tissue injury signaling

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In terms of renal effects, the evidence of SGLT2is in preventing kidney failure is more abundant than for GLP-1RAs, which showed a beneficial effect on albuminuria and not on hard kidney endpoints [3, 6, 8]